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About Jianchi Ding, M.Sc., Ph.D, HCLD
After graduation, he was awarded a post-doctoral fellowship from the Natural Science and Engineering Research Council of Canada to further continue his research at the Animal Biotechnology and Embryo Laboratory, University of Guelph. The University of Guelph is one of the leading centers in embryo research in North America. There, Dr. Ding spent more than two years studying rabbit primordial germ cells, bovine embryo development 'in vitro', and embryo freezing. He mastered the micromanipulation techniques of gametes and embryos while at Cambridge and Guelph. During the past 20 years, he has published more than 23 scientific research papers in peer-review journals and co-authored two major book chapters. He has presented more than 30 scientific reports at national and international meetings. His research findings have been frequently quoted by other authorities in the field. Dr. Ding joined OBFC in early 1996 and has successfully incorporated his research experience, especially in the area of micromanipulation, into our laboratory technology. He has also established an 'in vitro' co-culture system of embryos and patient's own endometrial cells which has improved the success rate in patients with at least one previously failed IVF cycle. Under his direction, our laboratory continues to maintain high success rates. Furthermore, Dr. Ding has recently been certified as a High Complexity Laboratory Director in the area of Embryology and Andrology after fulfillment of all the requirements and passing the examinations administered by the American Board of Bioanalysis. In 1997, Dr. Ding was appointed as a Visiting Professor at the Nanjing Medical University and helped in the establishment of the IVF center there. Dr. Ding has also been involved in the clinical and basic research studies at OBFC and the Institute for the Study and Treatment of Endometriosis. These studies produced interesting results leading to new hypotheses on the cause of endometriosis which indicate that women with endometriosis have reduced spontaneous programmed cell death (apoptosis) in their endometrial cells. This reduction of apoptosis may cause the increased viability of endometrial cells shed during the menstrual period, thus leading to their successful implantation outside the uterine cavity. | |
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